腰椎间盘髓核脱出重吸收的研究进展△

Research progress on resorption of prolapsed nucleus pulposus of lumbar intervertebral disc

摘要:

破裂型腰椎间盘突出症的发病机制是腰椎间盘髓核突出后突破后纵韧带并脱出甚至离开原椎间隙向上或向下游离， 此类型腰椎间盘突出症具有压迫程度严重、症状重的特点，并有发生神经损害的可能，一般多积极手术治疗。近年来愈来愈多的研究发现破裂型腰椎间盘突出症患者的髓核更容易发生重吸收，并且保守治疗相对安全，这为保守治疗破裂型腰椎间盘突出症提供了临床依据。本文就破裂型腰椎间盘突出症的临床特点、重吸收发生的机制、保守治疗的安全性及风险进行综述。

Abstract:

The pathogenesis of ruptured lumbar disc herniation is that the nucleus pulposus of the lumbar intervertebral disc break- through the posterior longitudinal ligament and prolapse or even leaves the original intervertebral space with up or down migration. This type of lumbar disc herniation has the characteristics of serious compression, severe symptoms and the possibility of nerve damage, usually treat- ed with surgery. In recent years with the in-depth study of clinical and imaging, it is found that the nucleus pulposus of ruptured lumbar disc herniation is more prone to be resorbed, and more suitable for conservative treatment, which provides a clinical basis for conservative treat- ment of ruptured lumbar disc herniation. This article reviews the clinical characteristics, mechanism of resorption, safety and risk of conser- vative treatment for ruptured lumbar disc herniation.